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LIFESPAN I UNIT 3 AND 4

TEST UNIT 3 AND 4

AB
LEFT TO RIGHT SHUNTBLOOD SHUNTED FROM AREA OF HIGH PRESSURE(LEFT SIDE) TO AN AREA OF LOWER PRESSURE (RIGHT SIDE)
RESULTS OF LEFT TO RIGHT SHUNTSACYANOTIC DEFECT, INCREASED PULMONARY WORKLOAD, CHF
PATENT DUCTUS ARTERIOSUS (PDA)FAILURE OF CLOSURE ALLOWSBLOOD TO SHUNT FROM THE HIGH PRESSURE AORTA TO THE LOWER PRESSURE PULMONARY ARTERY
ATRIAL SEPTAL DEFECTENABLES BLOOD TO FLOW FROM THE HIGH PRESSURE LEFT ATRIUM TO THE LOW PRESSURE RIGHT ATRIUM
VENTRICULAR SEPTAL DEFECTABNORMAL OPENING IN THE INTERVENTRICULAR SEPTUM, RESULTING IN THE FLOW OF BLOOD FROM THE HIGH PRESSURE LEFT VENTRICLE TO THE LOW PRESSURE RIGHT VENTRICLE
COARCTATION OF THE AORTA ( COA)NARROWING OF THE LUMEN OF THE AORTA, RESULTING IN INCREASED PRESSURE PROXIMAL TO THE DEFECT AND DECREASED PRESSURE DISTAL TO THE DEFECT
FOUR DEFECTS OF TETRALOGY OF FALLOT (TOF)1.PULMONARY STENOSIS 2.VENTRICULAR SEPTAL DEFECT 3.RIGHT VENTRICULAR HYPERTROPHY 4. OVERRIDING AORTA
TRANSPOSITION OF THE GREAT ARTERIESPULMONARY ARTERY ARISES FROM THE LEFT VENTRICLE, AND THE AORTA ARISES FROM THE RIGHT VENTRICLE
BLOOD ALWAYS FLOWS FROMHIGH PRESSURE TO LOW PRESSURE
SICKLE CELL ANEMIAABNORMAL FORM OF HEMOGLOBIN WITH THE ERYTHROCYTE
VASOOCCLUSIVE CRISISOBSTRUCTION OF THE SMLL BLOOD VESSELS OF THE HANDS AND FEET RESULTING IN EDEMA, IMPAIRED RANGE OF MOTION AND PAIN
MEDICAL MANAGEMENT VASOOCCLUSIVE CRISISPALLIATIVE-ANALGESICS, HYDRATION, OXYGEN
SEQUESTRATION CRISISBLOOD POOLS IN THE SPLEEN AND LIVER RESULTING IN HEPATOSPLENOMAGALY; CAN PRODUCE CARDIOVASCULAR COLLAPSE AND DEATH
MEDICAL MANAGEMENT SEQUESTRATION CRISISANALGESICS, VOLUME EXPANDERS, TRANSFUSIONS, SPLENECTOMY
APLASTIC CRISISCAUSED BY PREMATURE DESTRUCTION OF ERYTHROCYTES, RESULTING IN PROFOUND ANEMIA
MEDICAL MANAGEMENT APLASTIC CRISISTRANSFUSION
TYPES OF SICKLE CELL CRISESVASOOCCLUSIVE, SEQUESTRATION, APLASTIC
HEMOPHILIABLEEDING DISORDER INHERITED AS AN X-LINKED RECESSIVE DISORDER. TRANSMITTED BY FEMALE TO MALE OFFSPRING
HEMOPHILIA TYPE ADEFICIENCY OF CLOTTING FACTOR VIII
JUVENILE RHEUMATOID ARTHRITIS (JRA)CHRONIC INFLAMMATORY, AUTOIMMUNE DISEASE OF CONNECTIVE TISSUE
PEAK ONSET OF JRAAGES 2 TO 5 AND 9 TO 12
TYPES OF JRASYSTEMIC, POLYARTICULAR, PAUARTICULAR
POLYARTICULAR JRA>5 JOINTS AFFECTED
PAUARTICULAR<3 JOINTS AFFECTED
BRONCHIOLITISAN INFLAMMATION OF THE BRONCHIOLES THAT CAUSES PRODUCTION OF A THICK MUCUS THAT OCCLUDES BRONCHIOLE TUBES AND SMALL BRONCHI
RSVHIGHLY COMMUNICABLE, USUSALLY TRANSFERRED BY THE HANDS, COMMON CAUSE OF BRONCHIOLITIS
SIDSSUDDEN, UNEXPECTED, AND UNEXPLAINED DEATH OF A HEALTHY INFANT
PEAK INCIDENCE OF SIDS2 TO 4 MONTHS
SIDS OFTEN ASSOCIATED WITHPREMATURE BIRTH, LOW BIRTHWEIGHT, LOW aPGAR SCORES, MULTIPLE BIRTHS, CNS AND RESPIRATORY DYSFUNCTIONS
EPIGLOTTITISBACTERIAL FORM OF CROUP
TRIPOD POSITIONWHILE SUPPORTING BODY WITH HANDS, THE CHILD THRUSTS THE CHIN FORWARD AND OPENS THE MOUTH IN AN ATTEMPT TO WIDEN THE AIRWAY
INTERVENTIONS WITH EPIGLOTTITISADMINISTER ANALGESICS AND ANTIPYRETICS, COOL-MIST O2, PREPARE ET TUBE OR TRACH, ENCOURAGE HIB VACCINE
CROUPINFLAMMATION OF THE LARYNX, TRACHEA, AND BRONCHI, MAY BE VIRAL OR BACTERIAL
S/S CROUPSEAL BARK, BRASSY COUGH, INSPIRATORY STRIDOR, LABORED RESPIRATIONS, USE OF ACCESSORY MUSCLES FOR BREATHING
ASTHMAINCLUDES BRONCHOSPASM, EDEMA, AND INFLAMMATION OF THE BRONCHIAL AIRWAYS
S/S ASTHMADYSPNEA, EXPIRATORY WHEEZING, NONPRODUCTIVE COUGH, ORTHOPNEA, RESTLESSNESS
LONG CARE MANAGEMENT OF ASTHMAINSTRUCT THE CHILD TO RECOGNIZE EARLY SYMPTOMS OF AN ASTHMA ATTACK
CYSTIC FIBROSISMULTISYSTEM DISORDER AFFECTING EXOCRINE GLAND FUNCTION
SWEAT TESTSCREENING FOR CYSTIC FIBROSIS, AMOUNT OF NA AND CL IS MEASURED
POSTURAL DRAINAGEADMINISTER BEFORE MEALS
STEATORRHEABULKY, FOUL SMELLING STOOL
DEHYDRATIONMOST COMMON FLUID ELECTROLYTE IMBALANCE IN CHILDREN BECAUSE OF THEIR SIZE
CAUSES OF DEHYDRATIONDECREASED FLUID INTAKE, DIAPHORESIS, VOMITING, D
S/S DEHYDRATIONWEIGHT LOSS, DRY MUCOUS MEMBRANES, SUNKEN EYES, DEPRESSED FONTANELLES DECREASED URINE OUTPUT, INCREASED SPECIFIC GRAVITY, EXCESSIVE THIRST
DEHYDRATION INTERVENTIONSMONITOR WEIGHT, MONITOR I &O ADMINISTER IV FLUIDS AND ELECTROLYTE REPLACEMENTS IF PRESCRIBED ,
VOMITING CONCERNSRISK OF DEHYDRATION, LOSS OF FLUID AND ELECTROLYTES, AND THE DEVELOPMENT OF METABOLIC ALKALOSIS
DATA COLLECTION FOR VOMITINGFREQUENCY/AMOUNT
INTERVENTIONS FOR VOMITINGPATENT AIRWAY, I&O, KEEP NPO PROVIDE FLUID AS TOLERATED
CONCERNS WITH DIARRHEARISK OF DEHYDRATION, LOSS OF FLUID AND ELECTROLYTES, AND METABOLIC ACIDOSIS
DATA COLLECTION FOR DIARRHEAFREQUENC, CHARACTER, AMT.
CAUSES OF CLEFT LIP AND CLEFT PALATEGENETIC, HEREDITARY, AND ENVIRONMENTAL FACTORS, CHROMOSOME ABNORMALITIES, AND TERATOGENIC FACTORS
CLEFT LIP AND PALATEANOMALY THAT OCCURS AS A RESULT OF FAILURE OF SOFT TISSUE OR BONY STRUCTURE TO FUSE DURING EMBRYONIC DEVELOPMENT
AGE OF CLEFT LIP AND PALATE REPAIR6 MONTHS TO 2 YEARS BECAUSE EARLY CLOSURE FACILITATES SPEECH DEVELOPMENT
POST-OPERATIVE POSITION FOR CLEFT LIP/PALATEON SIDE LATERAL TO REPAIR OR ON BACK
GERBACKFLOW OF GASTRIC CONTENTS INTO THE ESOPHAGUS, AS A RESULT OF RELAXATION OR INCOMPETENCE OF THE LOWER ESOPHAGEAL OR CARDIAC SPHINCTER
COMPLICATIONS OF GERASPIRATION OF GASTRIC CONTENTS, ASPIRATION PNEUMONIA
TREATMENT FOR GERDIET, POSITIONING, MEDICATION, AND SURGERY
MEDICATIONS FOR GERANTACIDS, H2 RECEPTORS, PROKINETIC AGENTS
PYLORIC STENOSISHYPERTROPHY OF THE CIRCULAR MUSCLES OF THE PYLORUS CAUSES NARROWING OF THE PYLORIC CANAL BETWEEN THE STOMACH AND THE DUODENUM, SPASMS MOVE FOOD IN BOTH DIRECTIONS
S/S PYLORIC STENOSISVOMITING THAT PROGRESSES FROM MILD REGURGITATION TO PROJECTILE AND USUALLY OCCURS AFTER FEEDING
PYLOROTOMYFREDET-RAMSTEDT PROCEDURE, AN INCISION THROUGH THE MUSCLE FIBERS OF THE PYLORUS
POST-OPERATIVE PYLOROTOMYPOSITION IN HIGH FOWLER'S ON RIGHT SIDE AFTER FEEDING
HIRSCHSPRUNG'S DISEASEMEGACOLON ABSENCE OF GANGLION CELLS IN THE RECTUM AND UPWARD IN THE COLON, RESULTS IN MECHANICAL OBSTRUCTION FROM INADEQUETE MOTILITY IN AN INTESTIONAL SEGMENT
MEGACOLON TRAITSFAMILIAL CONGENITAL DEFECT, ASSOCIATED WITH DOWN SYNDROME, AND GENITAL URINARY ABNORMALITIES


MELLANY WEISS

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