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med surg cardio Ch. 53 nursing mgmt vascular

AB
PAD primary causeatherosclerosis
Atherosclerosis causessmoking, dm, hi chol, htn
common sites for PADaort iliac bifurcation; femoral bifurcations
byproducts of aneorbic metabolism in padlactic acid, pyruvate
claudicationischemic pain
claudication occurs most withexertion
claudication often occurs in ages60-70
claudication and PAD associated withCAD
ABI abnormal claudication likely.5-.9
ABI ishighest ankle / highest brachial systolic bp's
ABI normal levels.91 - 1.3
critical ischemia levels ABI<.49
calcified vessels, often in DM, innacuate results ABI is>1.3
arterial stealmax dilated hypoxic arterioles steal blood flow/oxygen from the nerves
paresthesiasnumbness/tingling sensation with PAD
critical limb ischemiano pulses, hypertrophied toenails, cool hairless legs, tissue atrophy, pareshesis
thse develop in ischemic legsulcers
Leriche syndromeimpotence that arises from PVD
when leg is dependent in PVD this occursrubor
tx of PVDsurgery, stents, etc after mappiing with arteriography
femoral artery branches intopopliteal & tibial arteries
acute arterial occlusion caused bythrombois, embolism, trauma
clinical manifestation of arterial occlusionpain, pulselessness, poikilothermic, pallor, paresthesias, paralysis
aneurysmsvenuous or arterial
abd. aortic aneurysmclassification by location & gross appearance
AAA becomes symptomatic at5 cm
AAA meidcal managmentmonitor every 6 mos.
AAA consider surgery6 cm
Type A aortic dissectioninvolves ascending aorta
Type B aortic dissectiondoesn't involve ascending aorta
clinical manisfestation of aortic dissectionabrucpt paun, tearung sensation, diminished sensatuion, htn
complications of aortic dissectioncardiac tamponade
Raynauds phenomenonvasospastic and obstructive problem
thromboangiitis obliterans akaBerger's disease
thromboangiitis obliterans isvasculitis in veins and arteries of young adults
thrombophlebitis riskshypercoaguability, injury to venous wall, venous stasis
thromboangiitis obliterans txnifedipine
thrombophlebitis pathophysiologyplts adhere to endothelium
thrombophlebitisred, warm, swollen, positive Homan's sign
thrombophlebitis testsvenouis duplex scanning; d dimer
thrombophlebitis txheparin, coumadin, monitor pt/tt
thrombophlebitis tx,surigcalvenous caval filter (Greenfield)
varicose veins due tolack of valvular competency
venuous insufficiencyblood pooling in periphery due to valves
thrombophlebitis nursingelevate legs, monitor for PE, reduce pain
venous stasis ulcerspoorly healing ulcers with irregular borders
varicose veins surgical txvein strippling, sclerosis
symptoms of venous stasisswollen limbs, thick brownish skin aroiund ankles, itchy skin arouind ankles
tx for venous stasis ulcreselevate legs, Unna boots, moist dressing
lymphedemaimpaired transcapillary fluid transport


Dr. Hyla Harvey
Marshall University Joan C. Edwards School of Medicine
Hurricane, WV

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