| A | B |
|---|
| right side of heart, pulmonary artery and veins | Pulmonary circulation |
| left side of heart, aorta, venous system, vena cava | Systemic circulation |
| Blood vessel diameter increases | Vasodilation |
| What part of the ANS causes decreased heart rate | PNS (parasympathetic nervous system) |
| Blood vessel diameter decreases | vasoconstriction |
| What part of the ANS causes increased heart rate, increased contractility, and vasoconstriction | SNS (sympathetic nervous system) |
| Ischemia | Decreased blood flow |
| Infarction | Tissue death |
| > 60 | HDL "good cholesterol" goal |
| Obesity, pregnancy, having a job where you stand all day | Some risk factors for varicose veins |
| Age, ethnicity, family history, gender | Non-modifiable risk factors for hypertension |
| Brown discoloration of skin, red in elevated position, cyanotic in dependent position, ulcer to ankle area | Manifestations of Chronic Venous Insufficiency |
| Hypertension can damage what organs? | Heart, brain, kidneys, eyes |
| <100 | LDL "bad cholesterol" goal |
| Diastolic Blood Pressure | Pressure in the arterial system when ventricles are relaxed. |
| > 200 mg/ dL | Hyperlipitdemia/ Hypercholesterolemia |
| Does chronic hypertension lead to vessel hypertrophy? | Yes |
| Risk factors: Men under 40 who smoke heavily | Thromboangiitis obliterans (Buerger's Disease) |
| These measure oxygen, carbon dioxide and hydrogen ion levels and can cause vasoconstriction | Chemoreceptors |
| Stasis of blood, vessel damage, hypercoagulability | Virchow's Triad |
| Blue, white, and red skin color are manifestations | Raynaud's Disease |
| Asymmetrical swelling, pain, redness, warmth | Manifestations of a DVT (deep vein thrombus) |
| Cholesterol deposit in tendons | Xanthomas |
| Risk of hemorrhage or death increases if the aneurysm is greater than | 5 mm |
| Deposit of lipids in intimal layer of blood vessel alonge with macrophages and cell debris | Foam cell |
| An abnormal drop in blood pressure associated with blood pressure is called | Orthostatic hypotension (postural hypotension) |
| Does collateral circulation happen quickly or over time | Over time |
| Bleeding between the layers of an arterial wall occurs with | Dissection of anuerysm |
| Air, bone marrow fat, irregular heartbeats, plaque, pregnancy complication (amniotic fluid) are all risk factors for the development of | Acute arterial occlusion |
| Systolic Blood Pressure | Pressure due to ejection of blood from the left ventricle |
| Feet pale when elevated and red when dependent | Atherosclerotic occlussive disease (PAD, PVD) |
| RAAS, ADH, Epinephrine | Humoral mechanisms of controlling blood pressure in the short term |
| Thick toe nails, risk of ulcer to great toe | Atherosclerotic occlussive disease (PAD, PVD) |
| manifestations of acute arterial occlusion | Sudden pain, pallor, polar, pulseless below, numbness, tingling, line of demarcation |
| What controls the long term regulation of blood pressure | The kidneys |
| Hypertension, atherosclerosis, degeneration of blood vessel are causes of | Aneurysms |
| stationary clot | Thrombus |
| An anserysm of this size can be palpable with pulsations | > 4 mm |
| True or false: Pulses will be weak or absent in atherosclerotic occlusive disease (PAD, PVD) | True - due to decreased or absent perfusion |
| traveling clot | Eemboli |
